L-type calcium channel-dependent inhibitory plasticity in the thalamus
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چکیده
منابع مشابه
L-type calcium channel-dependent inhibitory plasticity in the thalamus
Thalamocortical neurons integrate sensory and cortical activity and are regulated by input from inhibitory neurons in the thalamic reticular nucleus. Evidence suggests that during bursts of action potentials, dendritic calcium transients are seen throughout the dendritic tree of thalamocortical cells. Here, we review a recent study that suggests these calcium transients regulate inhibitory inpu...
متن کاملL - type Calcium Channel Dependent Inhibitory Plasticity in 1 the Thalamus
3 Sarah R Hulme and William M Connelly 4 5 1 School of Physiology and Pharmacology, School of Medical Sciences, University Walk, Bristol, BS8 6 1TD, United Kingdom. Email: [email protected] 7 8 2 Neuroscience Division, Cardiff School of Biosciences, The Sir Martin Evans Building, Museum 9 Avenue, Cardiff, CF10 3AX, United Kingdom. Phone: +44(0)29 208 79113 Email: 10 [email protected] ...
متن کاملNeuro Forum L-type calcium channel-dependent inhibitory plasticity in the thalamus
Hulme SR, Connelly WM. L-type calcium channel-dependent inhibitory plasticity in the thalamus. J Neurophysiol 112: 2037–2039, 2014. First published March 12, 2014; doi:10.1152/jn.00918.2013.— Thalamocortical neurons integrate sensory and cortical activity and are regulated by input from inhibitory neurons in the thalamic reticular nucleus. Evidence suggests that during bursts of action potentia...
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The perirhinal cortex of the temporal lobe is essential for the familiarity discrimination component of recognition memory. In view of the importance of changes in calcium ion concentration for synaptic plasticity, the present study examined the effects of L-type voltage-dependent calcium channel (VDCC) antagonism on rat perirhinal-based familiarity discrimination processes and plasticity inclu...
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The small GTPase Rap1 contributes to fear learning and cortico-amygdala plasticity by inhibiting glutamate release from cortical neurons, but mechanisms of this inhibition remain unknown. Conversely, L-type calcium channels (LTCCs) become involved in glutamate release after fear learning and LTP induction. Here, we show that Rap1 deletion in mouse primary cortical neurons increases synaptic ves...
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ژورنال
عنوان ژورنال: Journal of Neurophysiology
سال: 2014
ISSN: 0022-3077,1522-1598
DOI: 10.1152/jn.00918.2013